Gaining Weight with Crossfit and Why it Doesn't Matter. I have always been thin, so it was a bit of a surprise when I started gaining weight with Crossfit. I was born a preemie almost two months early, wore a size 4 in second grade, and didn’t start my period until just before I started high school. In high school I lived on macaroni, chicken nuggets, Taco Bell, hash browns, salads and fries smothered in ranch dressing. More recently, I ate chicken and pasta almost daily, Hungry Howie’s often, and Chinese or Mexican food for lunch more than once a week. I ate whatever I wanted whenever I wanted and not once did I think about my weight or size because I was genetically thin and that’s how I was going to be (as a reference, my dad at his heaviest was 5’1. September, 2. 01. It wasn’t until I started to lift weights early in 2. I actually started to see my weight go up. I went from forever 1.
While I spend most of my time educating people about sustainable approaches to getting a lean, strong, healthy physique., I do have an interest in the extreme of body. My friend needed to lose 25 pounds in a month without dieting. He had to lose at least 20 pounds in less than 30 days or he would be off the team. Inspired by Staci of Nerd. Fitness, I realized I was “skinny fat” before and first made the connection that the number on the scale has nothing to do with how you look. Massey Ferguson Tractors Discussion Forum Archived Messages. Return to Massey Ferguson Forum Main Page. Allis Chalmers: Avery: Bobcat: Case David Brown: Caterpillar. And so I continued to eat healthier and continue to lift weights, the weight continued to trend upwards. Gaining weight with Crossfit. My weight plateaued at 1. I started Crossfit, where it further increased, then was hanging around 1. But now with Eat to Perform, I have loosened up the paleo perspective which had caused me issues in the past, and my focus is on performance (edit: I wrote more about how healthy habits led me down an unhealthy path here). I won’t label it as “good” or “bad”, but increasing my calories and putting an emphasis on carbs has caused a fairly quick increase in weight in the past three months. I have a hard time putting my thoughts down at times because I have always been thin and cannot directly relate to so many women who struggle with weight or have these weight loss goals in mind. How can I explain to you that the number on the scale isn’t important when “society” tells you otherwise and when you have a “goal weight”? Who wants to listen to skinny me talk about eating whatever I want? I really have no idea if this will have any value to you, but I want to emphasize that the number is so insignificant. Some may think that the weight gain or additional mass could cause them to feel uncomfortable, others may see it as preventing them from participating in their ideal weight class, and still others are extremely content eating big and lifting big. Ultimately, it is about finding that place that you are most comfortable. One of the biggest shifts in my perception that I have had over the last couple years is that I don’t want to be the skinny girl. Increasing my bodyweight has allowed me to increase my lifts. This week alone, I PRed my front squat, bench press, push jerk, and deadlift. Do you want a small scale number? Or do you want more? I want bigger shoulders. I want to be able to push jerk my bodyweight. I want thicker thighs. I want to deadlift 3. I want that Crossfit Booty and want no risk of it ever leaving my side (well, backside.). I want to squat 2. I want to look strong, feel strong, be strong. Gaining weight has given me a bigger sense of self- confidence that just being “skinny” wasn’t. As of today, I am up 2. I first touched a barbell. I am up 1. 5lbs from when I started Crossfit, and up just about 1. I am sure that some amount of bodyfat has also gone in hand with the weight gain, but seeing bigger muscles, bigger “gainz,” and bigger lifts on the whiteboard has been a huge boost for me. The feeling of hitting PRs and my body doing things it never was able to do before, like a chest- to- bar pull up, is incomparable to anything else. While many of you may still use the scale as the ultimate measure and many of you prefer using working out and eating as a way to maintain a certain aesthetic look, I would challenge you to shift the focus to performance and see where it takes you. Your turn. Have you gained weight sine starting Crossfit? Would you prefer to be skinnier or stronger? How has your perception of your body changed since you started working out? Tagged as. crossfit. Anabolic steroid - Wikipedia. This article is about androgens as medications. For androgens as natural hormones, see Androgen. Anabolic steroids, also known more properly as anabolic- androgenic steroids (AAS). They are anabolic and increase protein within cells, especially in skeletal muscles. AAS also have varying degrees of androgenic and virilizing effects, including induction of the development and maintenance of masculinesecondary sexual characteristics such as the growth of the vocal cords and body hair. The word anabolic, referring to anabolism, comes from the Greek . The American College of Sports Medicine acknowledges that AAS, in the presence of adequate diet, can contribute to increases in body weight, often as lean mass increases and that the gains in muscular strength achieved through high- intensity exercise and proper diet can be additionally increased by the use of AAS in some individuals. Their use is referred to as doping and banned by most major sporting bodies. For many years, AAS have been by far the most detected doping substances in IOC- accredited laboratories. Testosterone is now nearly the only androgen used for this purpose and has been shown to increase height, weight, and fat- free mass in boys with delayed puberty. These sports include bodybuilding, weightlifting, shot put and other track and field, cycling, baseball, wrestling, mixed martial arts, boxing, football, and cricket. Such use is prohibited by the rules of the governing bodies of most sports. AAS use occurs among adolescents, especially by those participating in competitive sports. It has been suggested that the prevalence of use among high- school students in the U. S. Oral administration is the most convenient. Testosterone administered by mouth is rapidly absorbed, but it is largely converted to inactive metabolites, and only about 1/6 is available in active form. In order to be sufficiently active when given by mouth, testosterone derivatives are alkylated at the 1. This modification reduces the liver's ability to break down these compounds before they reach the systemic circulation. Testosterone can be administered parenterally, but it has more irregular prolonged absorption time and greater activity in muscle in enanthate, undecanoate, or cypionateester form. These derivatives are hydrolyzed to release free testosterone at the site of injection; absorption rate (and thus injection schedule) varies among different esters, but medical injections are normally done anywhere between semi- weekly to once every 1. A more frequent schedule may be desirable in order to maintain a more constant level of hormone in the system. In addition, because estered testosterone is dissolved in oil, intravenous injection has the potential to cause a dangerous embolism (clot) in the bloodstream. Transdermal patches (adhesive patches placed on the skin) may also be used to deliver a steady dose through the skin and into the bloodstream. Testosterone- containing creams and gels that are applied daily to the skin are also available, but absorption is inefficient (roughly 1. Individuals who are especially physically active and/or bathe often may not be good candidates, since the medication can be washed off and may take up to six hours to be fully absorbed. There is also the risk that an intimate partner or child may come in contact with the application site and inadvertently dose himself or herself; children and women are highly sensitive to testosterone and can suffer unintended masculinization and health effects, even from small doses. Injection is the most common method used by individuals administering AAS for non- medical purposes. Studies indicate that the anabolic properties of AAS are relatively similar despite the differences in pharmacokinetic principles such as first- pass metabolism. However, the orally available forms of AAS may cause liver damage in high doses. AAS were ranked 1. Long- term steroid abusers may develop symptoms of dependence and withdrawal on discontinuation of AAS. Recreational AAS use appears to be associated with a range of potentially prolonged psychiatric effects, including dependence syndromes, mood disorders, and progression to other forms of substance abuse, but the prevalence and severity of these various effects remains poorly understood. As a result, AAS users may get misdiagnosed by a psychiatrist not told about their habit. Case reports describe both hypomania and mania, along with irritability, elation, recklessness, racing thoughts and feelings of power and invincibility that did not meet the criteria for mania/hypomania. Compared with individuals that did not use steroids, young adult males that used AAS reported greater involvement in violent behaviors even after controlling for the effects of key demographic variables, previous violent behavior, and polydrug use. The drug response was highly variable. However: 8. 4% of subjects exhibited minimal psychiatric effects, 1. The mechanism of these variable reactions could not be explained by demographic, psychological, laboratory, or physiological measures. There have been anecdotal reports of depression and suicide in teenage steroid users. A 1. 99. 2 review found that AAS may both relieve and cause depression, and that cessation or diminished use of AAS may also result in depression, but called for additional studies due to disparate data. Most of these side- effects are dose- dependent, the most common being elevated blood pressure, especially in those with pre- existing hypertension. For example, AAS may prematurely stop the lengthening of bones (premature epiphyseal fusion through increased levels of estrogen metabolites), resulting in stunted growth. Other effects include, but are not limited to, accelerated bone maturation, increased frequency and duration of erections, and premature sexual development. AAS use in adolescence is also correlated with poorer attitudes related to health. Development of breast tissue in males, a condition called gynecomastia (which is usually caused by high levels of circulating estradiol), may arise because of increased conversion of testosterone to estradiol by the enzyme aromatase. This side- effect is temporary; the size of the testicles usually returns to normal within a few weeks of discontinuing AAS use as normal production of sperm resumes. Alteration of fertility and ovarian cysts can also occur in females. The kidney damage in the bodybuilders has similarities to that seen in morbidly obese patients, but appears to be even more severe. Water- soluble peptide hormones cannot penetrate the fatty cell membrane and only indirectly affect the nucleus of target cells through their interaction with the cell. However, as fat- soluble hormones, AAS are membrane- permeable and influence the nucleus of cells by direct action. The pharmacodynamic action of AAS begin when the exogenous hormone penetrates the membrane of the target cell and binds to an androgen receptor (AR) located in the cytoplasm of that cell. From there, the compound hormone- receptor diffuses into the nucleus, where it either alters the expression of genes. It has been hypothesized that this reduction in muscle breakdown may occur through AAS inhibiting the action of other steroid hormones called glucocorticoids that promote the breakdown of muscles. Through a number of mechanisms AAS stimulate the formation of muscle cells and hence cause an increase in the size of skeletal muscles, leading to increased strength. Depending on the length of use, the side effects of the steroid can be irreversible. Processes affected include pubertal growth, sebaceous gland oil production, and sexuality (especially in fetal development). Some examples of virilizing effects are growth of the clitoris in females and the penis in male children (the adult penis size does not change due to steroids. Men may develop an enlargement of breast tissue, known as gynecomastia, testicular atrophy, and a reduced sperm count. Compounds with a high ratio of androgenic to an anabolic effects are the drug of choice in androgen- replacement therapy (e. Determination of androgenic: anabolic ratio is typically performed in animal studies, which has led to the marketing of some compounds claimed to have anabolic activity with weak androgenic effects. This disassociation is less marked in humans, where all AAS have significant androgenic effects. The VP weight is an indicator of the androgenic effect, while the LA weight is an indicator of the anabolic effect. Two or more batches of rats are castrated and given no treatment and respectively some AAS of interest. The LA/VP ratio for an AAS is calculated as the ratio of LA/VP weight gains produced by the treatment with that compound using castrated but untreated rats as baseline: (LAc,t. The LA/VP weight gain ratio from rat experiments is not unitary for testosterone (typically 0. Animal studies also found that fat mass was reduced, but most studies in humans failed to elucidate significant fat mass decrements. The effects on lean body mass have been shown to be dose- dependent. Both muscle hypertrophy and the formation of new muscle fibers have been observed. The hydration of lean mass remains unaffected by AAS use, although small increments of blood volume cannot be ruled out. After drug withdrawal, the effects fade away slowly, but may persist for more than 6. Overall, the exercise where the most significant improvements were observed is the bench press. AR agonists are antigonadotropic . By suppressing endogenous testosterone levels and effectively replacing AR signaling in the body with that of the exogenous AAS, the myotrophic- androgenic ratio would be expected to be further increased, and this hence may be yet an additional mechanism contributing to the differences in myotrophic- androgenic ratio. In addition, some AAS, such as nandrolone, are also potent progestogens, and activation of the progesterone receptor is antigonadotropic similarly to activation of the AR. How To Get Ripped & Cut: Diet & Workout Guide. While I spend most of my time educating people about sustainable approaches to getting a lean, strong, healthy physique., I do have an interest in the extreme of body transformation, or how to get ripped. Why should you listen to me? I’ve achieved a ripped physique (photos on this page are of me) and helped guys with even “bad” genetics get ripped too. So not only does getting ripped take a ridiculous amount of effort and discipline, but the extreme is that it may not be healthy either. Let me give you an example: Let’s say there’s a guy Mike who weighs 1. Get Ripped Variable #2: Cardiovascular Activity. Similar to the amount of carbohydrates you eat, the amount of cardio you complete to lose the excess fat depends on your genetics. While I didn’t cover all the minutiae for how to get ripped, if you focus on the key elements I outline above, you will be able to achieve the very rare “ripped” physique. Want to follow a proven program to get ripped? Then check out my 1. Week Body Transformation Program.
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